Sekian Lama

Petang-petang macam nih baru de idea nak menulis... dah berbulan2 dah tak update hari nih datang seru nak mengupdate pulak... first2 tadi up grade kat gbr kat atas tu kasik cantik sikit.. hehehe cantik ker??? Sambil2 tambah gbr2 terbaru bersama En Petri huhuhu....

Semenjak menjadi suri rumah sepenuh masa nih de jelah nak di edit2 kat gbr2 lama mahupun yg baru tuh... selain tak menghilangkan skil mengedit saje kasik cun sikit gbr... maklumlah sekarang ramai lak org upload gbr2 instagram... oleh kerana aku tak berhand phone canggih make kene lah edit kat photoshop gbr ala2 instagram huhuhu.... agak2 ala2 retro2 sikit gbr tuh hehehe.... cun lah di mata ku kan.. kan...

Selain tuh mula dah melembut2kan tangan buat kraf2 tangan... sajek suke2 kan.... tapi memg dari dulu suka buat kraf2 tgn nih... bak kater kwn2 kreatif kater ko... hehehe ari tuh sibuk buat bunga guna felt, lepastu sekarang nih crepe paper pulak... tapi sebelum tuh aku duk menggulung2 tali guni kat surat khabar yg dibentuk bulat... pastu hias2 sikit dgn bunga plastik...kang dah edit2 sikit gbr aper kraf tangan yg di buat tu kasik cun kang aku upload la yea.... kot2 la de saper2 berminat nak tempah ker.. layanzzz leh gak tambah2 duit poket hehehe....

Dan... oleh kerana aku suka makan huhuhu de jelah nanti aku upload kan gambar2 juadah yg aku dah masak.... kot2 rajin kang aderlah resepi yg menyusul untuk tatapan semua.... aper salahnya berkongsi resepikan.. kan....

Okeh setakat nih dulu.... kain tak lipat lagi tuh... nak lipat kain lu kang En Petri balik bersepah lak rumah nih dgn kain tak berlipat... aper la suri rumah tapi rumah bersepah tak best la kan.... Ehhh ari nih tak yah masak mkn mlm yea....??? yuhuuuu rehat2 dulu maklum la mlm tadi baru kua wad lg.... hehehe.....

CIDP (Chronic Inflammatory Demyelinating Polyneuropathy)

CIDP (Chronic Inflammatory Demyelinating Polyneuropathy) is a rare disorder of the peripheral nerves characterized by gradually increasing weakness of the legs and, to a lesser extent, the arms.

It is the gradual onset as well as the chronic nature of CIDP that differentiates it from GBS. Fortunately, CIDP is even more rare than GBS. The incidence of new cases is estimated to be between 1.5 and 3.6 in a million people (compare to GBS: 1-2 in 100,000).

Like GBS, CIDP is caused by damage to the covering of the nerves, called myelin. It can start at any age and in both genders. Weakness occurs over two or more months.

Unlike GBS, CIDP is not self-limiting (with an end to the acute phase). Left untreated, 30% of CIDP patients will progress to wheelchair dependence. Early recognition and treatment can avoid a significant amount of disability.

	What causes CIDP? Current theory holds that the body’s immune system which normally protects it, perceives myelin as foreign and attacks it. Just what starts this process is not clear. Some patients are found to have abnormal proteins in their blood, and these may facilitate damage.
	How is CIDP diagnosed? Diagnosis of CIDP is based on the symptoms of the patient: difficulty walking which progressively worsens over a few months tingling or other abnormal sensations may also be experienced loss of reflexes, such as the knee and ankle jerk Tests may include: an electrical test, a nerve conduction velocity-electromyography study a spinal tap, to analyze cerebrospinal fluid blood and urine tests, including analysis of proteins
	How is CIDP treated? Treatment options are similar to GBS treatment, though will not require a lengthy hospital stay: Prednisone, similar to protective anti-inflammatory corticosteroids that are normally made by the body, may be used as an initial treatment for several reasons. It often improves strength, can be conveniently taken by mouth, and is inexpensive. Side effects can limit its use. High dose Intravenous Immune Globulins (IVIG), protective blood proteins obtained from healthy volunteers, can be readily given through an arm vein. Plasma Exchange (PE), or Plasmapheresis, is when some of the patient’s blood is removed and the blood cells returned without the liquid plasma portion of the patient’s blood. It may work by removing harmful antibodies contained in the plasma. Treatment of CIDP is somewhat of an art. If a patient shows good improvement with an initial treatment but experiences additional weakness, it may be repeated or another therapy may be tried. If treated early and aggressively, most CIDP patients respond well to therapy that can limit the damage to peripheral nerves and contribute to improved function and quality of life.
	Variants There are three general forms of CIDP: Progressive form extending over several years Recurrent form with multiple episodes that may be separated by months or years Monophasic form in which a single episode extends over one to three years without recurrence
	Living with CIDP Post-treatment life depends on whether the disease was caught early enough to benefit from treatment options. Patients respond in various ways. The gradual onset of CIDP can delay diagnosis by several months or even years, resulting in significant nerve damage that may take several courses of treatment before benefits are seen. The chronic nature of CIDP differentiates long-term care from GBS patients. Adjustments inside the home may need to be made to facilitate a return to normal life.

Temanku.....

Title temanku..... tapi bukanlah nak bercerita pasal kawan2 ku.... Beberapa bulan lepas aku dapat panggilan dari abg ku kat kampung, katnya ada sekor anak kucing dtg kerumah. Bulu kelabu bermata biru... Ya Allah gembiranya hati dgr berita tu.... maklum la mase tuh aku pun memang tengah mencari-cari anak kucing untuk di bela.... Biarpun pada mulanay berangan-angan nak anak kucing color oren, tapi kelabu pun kelabulah.... asalkan ada teman kala-kala berseorangan kat rumah maklumalh belum de cahaya mata lg hehehe..... Tapi risau gak, suke ke En Petri ngan kucing tu nanti... dia tak membantah aku nak bela kucing.... tapi tak pernah pulak tgk dia mesra-mesra ngn kucing.....

Apa pun sekarang kucing tuh dah duduk ngn kami dan rupanya En Petri gembira dengan kehadiran kucing tu.... luper lak aku bagi nama OGGY kuciong kami tuh sempena nama watak kucing dalam karton Oggy And The Cockroaches sebabnya rumah kami byk lipas... hehehe...

Kehadiran oggy sgt menceriakan kami, balik kg pun semorang suka kat oggy alhamdulillah.... Tapi kadang-kadang tu kalangkabut jugak la.... first day dia kat rumah dah kencing atas tilam.... geram ader tapi rase lawak lak.... dah malam2 basuk cadar huhuhu... tapi sekarang okey dah pandai nak membuang dalam kandang dia jek... tapi jadi kerja lah nak buang pulak ke tong sampah... belum sempat nak ajar membuang kat pasir, pasirnye pun tak belikan lagi hehehe....

Paling seronok dia suka main, tak kira waktu nak main jek... kadang-kadang dia siap bawak mainan dia tu ke kami suruh main ngn dialah tuh.... Banyak la benda-benda baru pasal kucing yg aku tau sejak kehadiran Oggy nih.... Selain suka main dia suka tgk tv huhuhu mcm tak caya lak.... kadang-kadang spi tertido tengok tv.... Kegemaranya tgk bola huhuh kang geram sgt spi dipanjat-panjat tv tuh hehehe...

Harap-harap Oggy akan jadik teman mami ngn dedi spi la kehujung nyawanya.... WE LOVE U OGGY..... muah... muah... muah....

My Best Friends......

What is Psoriasis?

Psoriasis (sore-EYE-ah-sis) is a medical condition that occurs when skin cells grow too quickly. Faulty signals in the immune system cause new skin cells to form in days rather than weeks. The body does not shed these excess skin cells, so the cells pile up on the surface of the skin and lesions form.

What are the signs and symptoms?
The lesions vary in appearance with the type of psoriasis. There are five types of psoriasis: Plaque, guttate, pustular, inverse, and erythrodermic. About 80% of people living with psoriasis have plaque (plak) psoriasis, also called “psoriasis vulgaris.” Plaque psoriasis causes patches of thick, scaly skin that may be white, silvery, or red. Called plaques (plax), these patches can develop anywhere on the skin. The most common areas to find plaques are the elbows, knees, lower back, and scalp.

Psoriasis also can affect the nails. About 50% of people who develop psoriasis see changes in their fingernails and/or toenails. If the nails begin to pull away from the nail bed or develop pitting, ridges, or a yellowish-orange color, this could be a sign of psoriatic (sore-EE-at-ic) arthritis. Without treatment, psoriatic arthritis can progress and become debilitating. It is important to see a dermatologist if nail changes begin or joint pain develops. Early treatment can prevent joint deterioration.

What causes psoriasis?
Psoriasis is not contagious. You cannot get psoriasis from touching someone who has psoriasis, swimming in the same pool, or even intimate contact. Psoriasis is much more complex.

So complex, in fact, scientists are still studying what happens when psoriasis develops. We know that the person’s immune system and genes play key roles. In studying the immune system, scientists discovered that when a person has psoriasis, the T cells (a type of white blood cell that fights unwanted invaders such as bacteria and viruses) mistakenly trigger a reaction in the skin cells. This is why you may hear psoriasis referred to as a “T cell-mediated disease.”

This reaction activates a series of events, causing new skin cells to form in days rather than weeks. The reason T cells trigger this reaction seems to lie in our DNA. People who develop psoriasis inherit genes that cause psoriasis. Unlike some autoimmune conditions, it appears that many genes are involved in psoriasis.

Scientists are still trying to identify all of the genes involved. One of the genes that has been identified is called PSORS1 (SORE-ESS-1). This is one of several genes that regulates how the immune system fights infection.

Scientists also have learned that not everyone who inherits genes for psoriasis gets psoriasis. For psoriasis to appear, it seems that a person must inherit the “right” mix of genes and be exposed to a trigger. Some common triggers are a stressful life event, skin injury, and having strep throat. Many people say that that their psoriasis first appeared after experiencing one of these. Triggers are not universal. What triggers psoriasis in one person may not cause psoriasis to develop in another.

 
Who gets psoriasis?
People worldwide develop psoriasis. In the United States, nearly 7.5 million people have psoriasis and about 150,000 new cases are diagnosed each year. Studies indicate that psoriasis develops about equally in males and females. Research also shows that Caucasians develop psoriasis more frequently than other races. A study conducted in the United States found the prevalence was 2.5% in Caucasians and 1.3% in African Americans.

A family history of psoriasis seems to increase the risk of developing psoriasis. It is important to know that a family history of psoriasis does not guarantee that someone will develop psoriasis.

 
When do people get psoriasis?
Psoriasis can begin at any age, from infancy through the golden years. There are, however, times when psoriasis is most likely to develop. Most people first see psoriasis between 15 and 30 years of age. About 75% develop psoriasis before they turn 40. Another common time for psoriasis to begin is between 50 and 60 years of age.

 
Does psoriasis affect quality of life?
For some people, psoriasis is a nuisance. Others find that psoriasis affects every aspect of their daily life. The unpredictable nature of psoriasis may be the reason. Psoriasis is a chronic (lifelong) medical condition. Some people have frequent flare-ups that occur weekly or monthly. Others have occasional flare-ups.

When psoriasis flares, it can cause severe itching and pain. Sometimes the skin cracks and bleeds. When trying to sleep, cracking and bleeding skin can wake a person frequently and cause sleep deprivation. A lack of sleep can make it difficult to focus at school or work. Sometimes a flare-up requires a visit to a dermatologist for additional treatment. Time must be taken from school or work to visit the doctor and get treatment.

These cycles of flare-ups and remissions often lead to feelings of sadness, despair, guilt and anger as well as low self-esteem. Depression is higher in people who have psoriasis than in the general population. Feelings of embarrassment also are common.

 
Knowledge is power
As psoriasis is a life-long condition, it is important to take an active role in managing it. Learning more about psoriasis, seeing a dermatologist to discuss treatment options, and developing a healthy lifestyle can help people live life to the fullest.

Picnic2......

Ari Jumaat malam Sabtu ari tu gi picnic ngn hubby tersayg.... Best..... Program yg tak dirancang pun.... patutnye mlm tu nak gi tgk wayg tapi hubby balik lambat lak.... tetiba dpt idea nak gi picnic cam zaman bercinta2 seblum kawen dulu hehehe..... menu yg senang & mengenyangkan..... sandwich sardin, baked beans, potato wedges (buatan sendiri), air soya bean hehehe....

Sampai dataran Shah Alam pun dah dekat kul 10 mlm.... terus bukk bekal & nyum.. nyum.. dengan kelaparanye huhuhu.... elok pulak angin pun bertiup sepoi2.... segar rasenye.... lain kali boleh ajak kwn2 datang join sekali... ramai2 ni meriah sikit... tambah pulak dengan anak2 dorang riuh... seronok rasenye....

Sambil2 mkn tgk budak2 main terhibur hati kami.... namun jauh disudut hatiku... kan best klu dapat main dengan anak2 kami sendiri... tak pelah.. belum de rezeki lg....

Habis mkn sempat lg kami merakamkan beberapa keping photo untuk kenangan2 masa2 akan datang.... hik.... mungkin mengambil gbr ke setiap destinasi yg kami pergi dah menjadi rutin.... sanggup aku sebelum kawen dulu beli kamera semuanya untuk merakamkan kenangan kami berdua... sambil2 tu aku menyimpan cita2 utk membeli kamera DLSR.... mungkin lambat lg tapi insyaallah klu ader rezeki... hehehe maklumlah klu guna kamera tu qualiti gbr lebih baik & cantik.... atau mungkin boleh buat bisnes dr tu... hehehe saper taukan....

Jam dah pukul 11 lebih... hubbyku ajak balik... esok dia nak keje pulak... hehehe aku jek cuti... aper pun mlm tu sgt indah utk dikenang.... saat2 menghabiskan masa ber2-2an... bergurau senda... bercerta zaman2 sblum kawen.. indahnya.. bahagianya....

Ya Allah panjangkan lah jodoh kami hingga ke akhir hayat kami...... sesungguhnya dia adalah anugerah terindah dalam hidup ku....

Apa pilihan yg ada......

Dihari yang penuh dengan pilihan..... namun setiap pilihan memerlukan pengorbana yang besar.... apa yang akan aku pilih.... semuanya demi masa depan ku, orang tersayg, keluarga dan mungkin orang-orang disekeliling.... ingin saja air mata ini menitis tatkala pilihan dibacakan.... namun... pernahkah aku menitiskan air mata ku sebelum ini kerananya??? belum lagi.... segalanya di pendam dalam hati.... tak dapat diluahkan..... tak dapat digambarkan.... pedihnya.. pilunya... ketabahan dan pengorbanan mungkin jawapan yang terbaik..... aku inginkan yang terbaik untuk diriku... tapi mampukah mereka menerima pilihanku ini... atau.... ku korbankan keinginan ku demi meraka???? sukarnya membuat pilihan..... Ya Allah.... dugaan apakah ini.....

Baru Sekarang....

Huh..... Sebulan br nak post something kat sini huhuhu....

Nothing yg baru and cerita2 yg best.... cuma meninggalkan Ramadhan dan Syawal dengan perasaan sedih and gembira. Sedih sebb belum tentu tahun depan dpt bertemu Ramadhan and Syawal lg.... Tapi klu dipanjangkan umur insyaallah...... Gembira kerana dpt menjalankan ibadah puasa and menyambut syawal bersama keluarga.....

Tahun ni best sebb semua adik beradikku balik and beraya bersama ehehehe.... dan raya keduaku sebagai seorg isteri.... hikhik....... alhamdulillah...

Lepas2 raya biarpun lemau2 jek nak bekerja kene la jugak gagahkan mencari rezeki..... Alhamdulillah rezeki lepas raya ader jek... ada lah 2-3 job free lance yg datang.....

Okeylah nak balik lu... kang rajin2 sambung kat umah... nak post gbr2 rayo nih....